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Disease Info Card
Tooth Attrition
Information about Tooth Attrition: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Tooth Attrition
Most recent studies have shown that Tooth Attrition shares some biological mechanisms with bruxism, dental-caries, dental-occlusion-traumatic, dentin-sensitivity, exanthema, fracture, gastroesophageal-reflux-disease, malocclusion, occlusal-wear-of-teeth, pain, periodontal-diseases, temporomandibular-joint-disorders, temporomandibular-joint-dysfunction-syndrome, tooth-abrasion, tooth-diseases, tooth-erosion, tooth-fractures, tooth-loss, tooth-wear.
Among the many pathways, these few ones have gauged particular interests from scientists studying Tooth Attrition, and have been seen in publications frequently: Aging, Ameloblast Differentiation, Amelogenesis, Bone Resorption, Dehiscence, Dentinogenesis, Feeding Behavior, Hypersensitivity, Localization, Mastication, Myelination, Ossification, Pathogenesis, Pigmentation, Reflex, Regeneration, Secretion, Senescence, Swimming, Tooth Eruption
Quite a number of genes have been found to play important roles in Tooth Attrition, such as ARHGAP32, CALM1, CALM2, CALM3, CAMKMT, DMP1, DSPP, EXOSC10, INPP5D, ISYNA1, KRIT1, RNF130, SLC27A4, SPP1, SS18L1, TNC, TNFSF14, TTN. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.