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Disease Info Card
Temporomandibular Joint Dysfunction Syndrome
Information about Temporomandibular Joint Dysfunction Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Temporomandibular Joint Dysfunction Syndrome
Most recent studies have shown that Temporomandibular Joint Dysfunction Syndrome shares some biological mechanisms with abnormal-degeneration, ankylosis, arthralgia, arthritis, arthropathy, bruxism, degenerative-polyarthritis, dental-occlusion-traumatic, dislocations, facial-pain, headache, inflammation, interosseous-desmitis, malocclusion, myeloproliferative-syndrome-transient, pain, soft-tissue-rheumatism, sore-to-touch, temporomandibular-joint-disorders.
Among the many pathways, these few ones have gauged particular interests from scientists studying Temporomandibular Joint Dysfunction Syndrome, and have been seen in publications frequently: Aging, Angiogenesis, Bone Resorption, Cell Proliferation, Endochondral Ossification, Hypersensitivity, Inflammatory Response, Innervation, Localization, Mastication, Muscle Atrophy, Muscle Contraction, Ossification, Pathogenesis, Proestrus, Reflex, Regeneration, Sensitization, Translation, Transport
Quite a number of genes have been found to play important roles in Temporomandibular Joint Dysfunction Syndrome, such as ACAT1, CALCA, CEP70, ESR1, FUT3, HPS4, IL1B, IL6, NOS2, NPY, PPP5C, PPT1, SLC25A5, TAC1, TFF2, TNF, TTN, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.