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Information about Syndactyly Of Toes With Fusion Of Bones: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Syndactyly Of Toes With Fusion Of Bones shares some biological mechanisms with carcinogenesis, carcinoma, chromosomal-deletion, cicatrix, extramedullary-hematopoiesis-(disorder), hyperplasia, limb-deformities-congenital, malignant-neoplasms, malignant-tumor-of-cervix, neoplasms, obesity, polydactyly, precancerous-conditions, radiation-chimera-disorder, rodent-diseases, sarcoma, syndactyly, thymus-hyperplasia, uterine-cervical-neoplasm.
Among the many pathways, these few ones have gauged particular interests from scientists studying Syndactyly Of Toes With Fusion Of Bones, and have been seen in publications frequently: Cell Cycle, Cell Cycle Arrest, Cell Death, Cell Division, Cell Maturation, Cell Migration, Cell Proliferation, Endocytosis, Hindlimb Morphogenesis, Induction Of Apoptosis, Limb Development, Limb Morphogenesis, Localization, Neural Tube Closure, Nuclear Export, Programmed Cell Death, Regulation Of Programmed Cell Death, Somitogenesis, Spermatogenesis, Tropism
Quite a number of genes have been found to play important roles in Syndactyly Of Toes With Fusion Of Bones, such as AHSA1, AKTIP, BMP4, EGF, EGFR, FTO, HOOK3, IRX3, IRX5, IRX6, MID1, NOLC1, PARP1, RBL2, RPGRIP1L, SHH. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.