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Disease Info Card
Polydactyly
Information about Polydactyly: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Polydactyly
Most recent studies have shown that Polydactyly shares some biological mechanisms with bardet-biedl-syndrome, cleft-lip, cleft-palate, congenital-abnormality, congenital-absence, congenital-cerebral-hernia, congenital-foot-deformity, congenital-hand-deformities, congenital-heart-defects, dwarfism, dysplasia, hydrocephalus, hypoplasia, limb-deformities-congenital, meckel-gruber-syndrome, obesity, radial-polydactyly, retinitis-pigmentosa, syndactyly.
Among the many pathways, these few ones have gauged particular interests from scientists studying Polydactyly, and have been seen in publications frequently: Bone Development, Cell Death, Cell Proliferation, Intraflagellar Transport, Limb Development, Limb Morphogenesis, Localization, Mating, Neural Tube Closure, Neural Tube Patterning, Ossification, Pathogenesis, Pigmentation, Programmed Cell Death, Regeneration, Segmentation, Translation, Transport, Transposition, Tube Closure
Quite a number of genes have been found to play important roles in Polydactyly, such as BBS1, BBS2, BBS4, BMP4, EVC, FGF4, FGF8, GLI1, GLI3, HOXD13, LMBR1, MKKS, MKS1, PTCH1, SHH, TMEM67, ZP2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.