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Disease Info Card
Sialic Acid Storage Disease, Finnish Type (disorder)
Information about Sialic Acid Storage Disease, Finnish Type (disorder): characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Sialic Acid Storage Disease, Finnish Type (disorder)
Most recent studies have shown that Sialic Acid Storage Disease, Finnish Type (disorder) shares some biological mechanisms with ataxia, atrophy, cystinosis, developmental-delay-(disorder), inborn-errors-of-metabolism, infantile-sialic-acid-storage-disease, lysosomal-storage-diseases, mucolipidoses, muscle-hypotonia, neurodegenerative-disorders, sialuria, storage-disease, type-i-mucolipidosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Sialic Acid Storage Disease, Finnish Type (disorder), and have been seen in publications frequently: Amino Acid Transport, Aspartate Transport, Autophagy, Endosomal Transport, Excretion, Exocytosis, Glycosylation, Intracellular Transport, Localization, Lysosomal Transport, Methylation, Myelination, Oligodendrocyte Development, Pathogenesis, Rna Interference, Secretion, Sialic Acid Transport, Symport, Transport
Quite a number of genes have been found to play important roles in Sialic Acid Storage Disease, Finnish Type (disorder), such as ARHGAP4, CD36, CP, CSF2, CTLA4, CTNS, HLA-DQA1, LAMC2, LAMP3, MTSS1, NOD2, PLA2G15, PRM1, RANGAP1, SCARB2, SLC16A1, SLC16A7, SLC17A5. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.