Disease Info Card

Osteoporotic Fractures

Information about Osteoporotic Fractures: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Osteoporotic Fractures

Most recent studies have shown that Osteoporotic Fractures shares some biological mechanisms with arthritis, back-pain, bone-diseases, cardiovascular-diseases, degenerative-polyarthritis, diabetes-mellitus, femoral-fractures, fracture, fractures-compression, hip-fractures, malignant-neoplasm-of-breast, malignant-neoplasms, metabolic-bone-disorder, osteopenia, osteoporosis, osteoporosis-postmenopausal, pain, pathological-fracture, spinal-fractures, vitamin-d-deficiency.

Among the many pathways, these few ones have gauged particular interests from scientists studying Osteoporotic Fractures, and have been seen in publications frequently: Aging, Bone Development, Bone Mineralization, Bone Remodeling, Bone Resorption, Cognition, Excretion, Lactation, Localization, Menarche, Menopause, Menstruation, Ossification, Osteoblast Differentiation, Osteoclast Differentiation, Pathogenesis, Regeneration, Secretion, Translation, Transport

Quite a number of genes have been found to play important roles in Osteoporotic Fractures, such as BEST1, BGLAP, CALCA, DMD, ESR1, FN1, GRIP1, IGF1, NDUFB6, PTH, PTRH1, RANGAP1, SHBG, SLC17A5, SP1, TNF, TNFRSF11B, TNFSF11. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.