Disease Info Card

Pathological Fracture

Information about Pathological Fracture: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Pathological Fracture

Most recent studies have shown that Pathological Fracture shares some biological mechanisms with bone-diseases, bone-neoplasms, femoral-fractures, femoral-neck-fractures, fracture, hip-fractures, humeral-fractures, malignant-neoplasms, malignant-paraganglionic-neoplasm, metastatic-malignant-neoplasm-to-the-bone, multiple-myeloma, neoplasm-metastasis, neoplasms, osteopenia, osteoporosis, osteoporosis-postmenopausal, osteoporotic-fractures, pain, spinal-fractures, stress-fractures.

Among the many pathways, these few ones have gauged particular interests from scientists studying Pathological Fracture, and have been seen in publications frequently: Aging, Angiogenesis, Bone Mineralization, Bone Remodeling, Bone Resorption, Cell Proliferation, Coagulation, Dentinogenesis, Enucleation, Excretion, Lactation, Localization, Menopause, Ossification, Osteoblast Differentiation, Pathogenesis, Regeneration, Secretion, Transport, Wound Healing

Quite a number of genes have been found to play important roles in Pathological Fracture, such as BEST1, BGLAP, CALCA, DMD, ESR1, HHIP, KLK3, PTH, PTRH1, REG3A, RPL29, SS18L1, ST13, TNFRSF11A, TNFRSF11B, TNFSF11. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.