Disease Info Card

Osteoporosis, Postmenopausal

Information about Osteoporosis, Postmenopausal: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Osteoporosis, Postmenopausal

Most recent studies have shown that Osteoporosis, Postmenopausal shares some biological mechanisms with back-pain, bone-diseases, cardiovascular-diseases, coronary-heart-disease, endometrial-polyp, fracture, heart-diseases, hip-fractures, hot-flushes, malignant-neoplasm-of-breast, malignant-neoplasms, mammary-neoplasms, metabolic-bone-disorder, oestrogen-deficiency, osteopenia, osteoporosis, osteoporotic-fractures, pain, pathological-fracture, spinal-fractures.

Among the many pathways, these few ones have gauged particular interests from scientists studying Osteoporosis, Postmenopausal, and have been seen in publications frequently: Aging, Bone Mineralization, Bone Remodeling, Bone Resorption, Cell Proliferation, Coagulation, Cognition, Cytokine Production, Excretion, Glomerular Filtration, Intestinal Absorption, Lactation, Menarche, Menopause, Menstruation, Osteoblast Differentiation, Osteoclast Differentiation, Pathogenesis, Secretion, Transport

Quite a number of genes have been found to play important roles in Osteoporosis, Postmenopausal, such as ALPP, ASRGL1, ATRNL1, BEST1, BGLAP, CALCA, DMD, ESR1, IGF1, IL6, PTH, PTRH1, RANGAP1, SLC17A5, SLPI, TNF, TNFRSF11A, TNFRSF11B, TNFSF11. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.