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Disease Info Card
Optic Atrophy, Hereditary, Leber
Information about Optic Atrophy, Hereditary, Leber: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Optic Atrophy, Hereditary, Leber
Most recent studies have shown that Optic Atrophy, Hereditary, Leber shares some biological mechanisms with amaurosis, atrophy, blind-vision, disorder-of-the-optic-nerve, hereditary-diseases, leber-congenital-amaurosis, mitochondrial-diseases, multiple-sclerosis, neuritis, optic-atrophies-hereditary, optic-atrophy, optic-atrophy-autosomal-dominant, optic-neuritis, retinal-diseases, retinitis-pigmentosa, sclerosis, visual-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Optic Atrophy, Hereditary, Leber, and have been seen in publications frequently: Aging, Cell Cycle, Cell Death, Cell Differentiation, Cell Growth, Cellular Respiration, Dna Repair, Electron Transport, Electron Transport Chain, Hypersensitivity, Localization, Myelination, Oxidative Phosphorylation, Pathogenesis, Phototransduction, Reflex, Regeneration, Response To Oxidative Stress, Translation, Transport
Quite a number of genes have been found to play important roles in Optic Atrophy, Hereditary, Leber, such as AIPL1, CLTA, CRB1, CYCS, ERG, GUCY2D, IVNS1ABP, KCNH2, MED12, MT-ND6, OPA1, PTPRC, RPE65, RPGRIP1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.