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Disease Info Card
Oculomotor Apraxia
Information about Oculomotor Apraxia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Oculomotor Apraxia
Most recent studies have shown that Oculomotor Apraxia shares some biological mechanisms with apraxia-oculomotor-cogan-type, apraxias, ataxia, ataxia-telangiectasia, atrophy, cerebellar-ataxia, familial-aplasia-of-the-vermis, friedreich-ataxia, gaucher-disease, motor-apraxia, muscle-hypotonia, neurodegenerative-disorders, nystagmus, ocular-motility-disorders, oculomotor-nerve-paralysis, ophthalmoplegia, peripheral-neuropathy, spinocerebellar-degeneration, telangiectasis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Oculomotor Apraxia, and have been seen in publications frequently: Aging, Brainstem Development, Cell Adhesion, Cell Cycle, Chromosome Breakage, Dna Ligation, Dna Repair, Dna Replication, Double-strand Break Repair, Hypersensitivity, Localization, Muscle Atrophy, Myelination, Neurogenesis, Pathogenesis, Pigmentation, Reflex, Response To Oxidative Stress, Rna Processing, Single Strand Break Repair
Quite a number of genes have been found to play important roles in Oculomotor Apraxia, such as AFP, AHI1, ALB, APTX, ATM, FHIT, FXN, GBA, NPHP1, NPHP4, PARP1, SETX, TRIM26, TTPA, XRCC1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.