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Disease Info Card
Oculomotor Nerve Paralysis
Information about Oculomotor Nerve Paralysis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Oculomotor Nerve Paralysis
Most recent studies have shown that Oculomotor Nerve Paralysis shares some biological mechanisms with abducens-nerve-diseases, aneurysm, blepharoptosis, cranial-nerve-diseases, cranial-nerve-palsies, diplopia, headache, hemorrhage, infarction, intracranial-aneurysm, neoplasms, nerve-paralysis, ocular-motility-disorders, ophthalmoplegia, pain, paresis, pituitary-diseases, strabismus, subarachnoid-hemorrhage.
Among the many pathways, these few ones have gauged particular interests from scientists studying Oculomotor Nerve Paralysis, and have been seen in publications frequently: Aging, Cell Adhesion, Cell Cycle, Cell Cycle Checkpoint, Dna Repair, Double-strand Break Repair, Flight, Hypersensitivity, Immune Response, Innervation, Localization, Muscle Contraction, Ossification, Pathogenesis, Pigmentation, Reflex, Regeneration, Response To Oxidative Stress, Transposition, Vestibular Reflex
Quite a number of genes have been found to play important roles in Oculomotor Nerve Paralysis, such as AFP, APTX, AURKA, CLIP1, CLIP2, CSF2, FLVCR1, FUT3, ICA, KIF21A, KLK3, LAMC2, POMC, PRL, ROBO3, SETX, TRIM26. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.