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Disease Info Card
Myopathies, Nemaline
Information about Myopathies, Nemaline: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Myopathies, Nemaline
Most recent studies have shown that Myopathies, Nemaline shares some biological mechanisms with arthrogryposis, atrophy, cardiomyopathies, central-core-myopathy-(disorder), congenital-myopathy-(disorder), congenital-structural-myopathy, dystrophy, hypertrophy, malignant-hyperpyrexia-due-to-anesthesia, malignant-paraganglionic-neoplasm, muscle-hypotonia, muscle-weakness, muscular-atrophy, muscular-dystrophy, myopathy, neuromuscular-diseases, respiratory-failure, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Myopathies, Nemaline, and have been seen in publications frequently: Aging, Cardiac Muscle Contraction, Cell Death, Cell Migration, Fertilization, Flight, Hypersensitivity, Innervation, Localization, Muscle Atrophy, Muscle Cell Differentiation, Muscle Contraction, Myofibril Assembly, Nuclear Export, Pathogenesis, Reflex, Regeneration, Regulation Of Muscle Contraction, Regulation Of Striated Muscle Contraction, Striated Muscle Contraction
Quite a number of genes have been found to play important roles in Myopathies, Nemaline, such as ACTA1, CFL2, DES, DMD, MCL1, MTM1, MYH14, MYOT, NEB, RUNX2, RYR1, THBD, TNNT1, TPM1, TPM2, TPM3, TTN, VIM. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.