Cardiac Muscle Contraction

Overview of Cardiac Muscle Contraction

Most recent studies have shown that Cardiac Muscle Contraction shares some biological mechanisms with aging, cell-activation, cell-migration, electron-transport, electron-transport-chain, histone-modification, hyperphosphorylation, localization, membrane-depolarization, muscle-contraction, oxidative-phosphorylation, pathogenesis, protein-phosphorylation, protein-targeting, proteolysis, regulation-of-muscle-contraction, skeletal-muscle-contraction, striated-muscle-contraction, transport.

Among the many pathways, these few ones have gauged particular interests from scientists studying Cardiac Muscle Contraction, and have been seen in publications frequently: aging, cell-activation, cell-migration, electron-transport, electron-transport-chain, histone-modification, hyperphosphorylation, localization, membrane-depolarization, muscle-contraction, oxidative-phosphorylation, pathogenesis, protein-phosphorylation, protein-targeting, proteolysis, regulation-of-muscle-contraction, skeletal-muscle-contraction, striated-muscle-contraction, transport

Quite a number of genes have been found to play important roles in Cardiac Muscle Contraction, such as CALM1, CALM2, CALM3, FTH1, INS, LDLR, Myh14, RYR1, RYR2, SCTR, SNCG, TNC, TNNC1, TNNI3, TNNT1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this pathway. Plesae stay updated.

Cardiac Muscle Contraction Related Genes

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Diseases Related to Cardiac Muscle Contraction

This information is being compiled and will come in a future update

Related pathways