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Disease Info Card
Microvillous Inclusion Disease
Information about Microvillous Inclusion Disease: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Microvillous Inclusion Disease
Most recent studies have shown that Microvillous Inclusion Disease shares some biological mechanisms with atrophy, autoimmune-reaction, celiac-disease, chronic-diarrhea, cytomegalovirus-infections, diarrhea, diarrhea-watery, dysplasia, enteritis, infantile-diarrhea, intestinal-diseases, malabsorption-syndrome, mucolipidoses, protracted-diarrhea, pseudo-obstruction, secretory-diarrhea, severe-diarrhea, systemic-infection.
Among the many pathways, these few ones have gauged particular interests from scientists studying Microvillous Inclusion Disease, and have been seen in publications frequently: Apical Protein Localization, Cell Adhesion, Cell Development, Cell Division, Cell Proliferation, Cytokinesis, Dehiscence, Endocytosis, Endosome Organization, Epithelial Cell Proliferation, Excretion, Exocytosis, Glycosylation, Ion Transport, Localization, Pathogenesis, Protein Transport, Secretion, Stem Cell Division, Transport
Quite a number of genes have been found to play important roles in Microvillous Inclusion Disease, such as ACHE, CEACAM5, CEACAM7, EGF, FOXP3, GLB1, HPN, LCT, MME, MYO5B, PSG2, RAB8A, RANGAP1, SLC17A5, SLC9A3, SST, USB1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.