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Disease Info Card
Pseudo-obstruction
Information about Pseudo-obstruction: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Pseudo-obstruction
Most recent studies have shown that Pseudo-obstruction shares some biological mechanisms with abdominal-pain, colonic-diseases, colonic-pseudo-obstruction, constipation, diarrhea, diffuse-scleroderma, gastrointestinal-diseases, hirschsprung-disease, ileus, intestinal-diseases, intestinal-obstruction, intestinal-pseudo-obstruction, myopathy, nervousness, ogilvie-syndrome, pain, pathological-dilatation, vomiting.
Among the many pathways, these few ones have gauged particular interests from scientists studying Pseudo-obstruction, and have been seen in publications frequently: Acid Secretion, Cell Proliferation, Coagulation, Defecation, Excretion, Fermentation, Gastric Emptying, Gastric Motility, Hypersensitivity, Immune Response, Innervation, Intestinal Absorption, Muscle Atrophy, Oxidative Phosphorylation, Pathogenesis, Peristalsis, Reflex, Secretion, Smooth Muscle Atrophy, Transport
Quite a number of genes have been found to play important roles in Pseudo-obstruction, such as ACHE, ALB, DES, EDNRB, GNAI1, HPN, IL2, KIT, LCN1, MLN, NOS1, SS18L1, SST, TAF9, TEAD1, TLX2, TMPO, TYMP, USB1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.