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Disease Info Card
Microdontia
Information about Microdontia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Microdontia
Most recent studies have shown that Microdontia shares some biological mechanisms with aplasia-nos, cleft-palate, congenital-absence, dens-in-dente, dental-enamel-hypoplasia, developmental-absence-of-tooth, dwarfism, dysplasia, ectodermal-dysplasia, exanthema, hypodontia, hypoplasia, macrodontia, malocclusion, neoplasms, tooth-abnormalities, tooth-loss, tooth-supernumerary.
Among the many pathways, these few ones have gauged particular interests from scientists studying Microdontia, and have been seen in publications frequently: Amelogenesis, Bone Remodeling, Cell Cycle, Cell Cycle Arrest, Cell Differentiation, Cell Proliferation, Dentinogenesis, Ear Development, Germination, Inner Ear Development, Keratinization, Localization, Mating, Odontogenesis, Root Development, Secretion, Tooth Eruption, Transport, Transposition, Wound Healing
Quite a number of genes have been found to play important roles in Microdontia, such as ADAMTS2, BMP4, CDKN1A, CNTLN, FGF3, FOLR1, GJA1, IKBKG, IRF6, PCNT, PITX2, RUNX2, SMOC2, TBCE, TMCO1, TWIST1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.