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Disease Info Card
Dental Enamel Hypoplasia
Information about Dental Enamel Hypoplasia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Dental Enamel Hypoplasia
Most recent studies have shown that Dental Enamel Hypoplasia shares some biological mechanisms with amelogenesis-imperfecta, celiac-disease, congenital-abnormality, dental-caries, dental-fluorosis-acquired, developmental-absence-of-tooth, dysplasia, exanthema, gingivitis, hypodontia, hypoplasia, malocclusion, mouth-diseases, oral-manifestations, periodontal-diseases, physiological-stress, syndactyly, tooth-abnormalities, tooth-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying Dental Enamel Hypoplasia, and have been seen in publications frequently: Ameloblast Differentiation, Amelogenesis, Cell Adhesion, Cell Proliferation, Dentin Mineralization, Dentinogenesis, Enamel Mineralization, Hypersensitivity, Immune Response, Localization, Mating, Odontogenesis, Ossification, Pathogenesis, Pigmentation, Root Development, Secretion, Segmentation, Tooth Eruption, Transport
Quite a number of genes have been found to play important roles in Dental Enamel Hypoplasia, such as AIRE, ALB, AMBN, AMELX, COL17A1, DLX3, DSPP, ENAM, F3, GJA1, MMP20, MMP25, PTH, SS18L1, TDO2, TF, TUFT1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.