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Disease Info Card
Knocked Out
Information about Knocked Out: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Knocked Out
Most recent studies have shown that Knocked Out shares some biological mechanisms with alzheimers-disease, atrophy, autoimmune-reaction, brain-injuries, carcinoma, cell-transformation-neoplastic, craniocerebral-trauma, diabetes-mellitus, fibrosis, hypertrophy, infective-disorder, inflammation, inflammatory-response, kidney-diseases, malignant-neoplasms, neoplasms, nerve-degeneration, nervousness, tissue-adhesions, tuberculosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Knocked Out, and have been seen in publications frequently: Aging, Angiogenesis, Cell Cycle, Cell Cycle Arrest, Cell Death, Cell Differentiation, Cell Growth, Cell Proliferation, Dna Repair, Fermentation, Immune Response, Inflammatory Response, Localization, Mating, Pathogenesis, Rna Interference, Secretion, Translation, Transport, Virulence
Quite a number of genes have been found to play important roles in Knocked Out, such as APP, AR, CALM1, CALM2, CAT, CDKN1A, CDKN2A, CFTR, INS, JUN, MAPK1, MTOR, MYC, NFKB1, NOS2, PMEL, PRNP, TNF, TP53, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.