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Disease Info Card
Fuchs Endothelial Dystrophy
Information about Fuchs Endothelial Dystrophy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Fuchs Endothelial Dystrophy
Most recent studies have shown that Fuchs Endothelial Dystrophy shares some biological mechanisms with cataract, chandler-syndrome, cicatrix, corneal-diseases, corneal-edema, corneal-guttata, dislocations, dystrophy, edema, glaucoma, graft-failure, hereditary-corneal-dystrophy, intraocular-pressure-disorder, keratitis, refractive-errors.
Among the many pathways, these few ones have gauged particular interests from scientists studying Fuchs Endothelial Dystrophy, and have been seen in publications frequently: Aging, Cell Adhesion, Cell Death, Cell Growth, Cell Migration, Dehiscence, Endothelial Cell Migration, Immune Response, Innervation, Localization, Pathogenesis, Pigmentation, Programmed Cell Death, Reflex, Regeneration, Reverse Transcription, Secretion, Transport, Vasculogenesis, Wound Healing
Quite a number of genes have been found to play important roles in Fuchs Endothelial Dystrophy, such as A4GALT, ASAH1, CLU, COL8A2, DMPK, ECD, PBK, PYCARD, RANGAP1, SERINC3, SLC17A5, SLC4A11, SRI, TCF4, TCF7L2, ZEB1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.