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- Table of Contents
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Disease Info Card
Impaired Healing
Information about Impaired Healing: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Impaired Healing
Most recent studies have shown that Impaired Healing shares some biological mechanisms with complications-of-diabetes-mellitus, diabetes-mellitus, diabetes-mellitus-experimental, diabetic-foot, fracture, hemorrhage, infective-disorder, inflammation, inflammatory-response, ischemia, malignant-neoplasms, malnutrition, neoplasms, pain, pathologic-neovascularization, tissue-adhesions, tumor-angiogenesis, ulcer, wound-infection.
Among the many pathways, these few ones have gauged particular interests from scientists studying Impaired Healing, and have been seen in publications frequently: Aging, Angiogenesis, Cell Cycle, Cell Migration, Cell Proliferation, Chemotaxis, Coagulation, Dehiscence, Fibroblast Migration, Fibroblast Proliferation, Immune Response, Inflammatory Response, Localization, Pathogenesis, Regeneration, Response To Wounding, Secretion, Senescence, Tissue Remodeling, Wound Healing
Quite a number of genes have been found to play important roles in Impaired Healing, such as ALB, EGF, FGF2, FGF7, FN1, IGF1, IL6, INS, ITGB2, MAPK1, MMP2, MMP9, MTOR, NOS2, PECAM1, PLG, TGFB1, TNF, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.