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Disease Info Card
Wound Infection
Information about Wound Infection: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Wound Infection
Most recent studies have shown that Wound Infection shares some biological mechanisms with abscess, bacterial-infections, cross-infection, fracture, hematoma, hemorrhage, hernia, infective-disorder, malignant-neoplasms, neoplasms, pain, pathologic-fistula, pneumonia, staphylococcal-infections, surgical-wound, surgical-wound-infection, systemic-infection, ulcer, urinary-tract-infection.
Among the many pathways, these few ones have gauged particular interests from scientists studying Wound Infection, and have been seen in publications frequently: Angiogenesis, Coagulation, Dehiscence, Drug Resistance, Excretion, Gastric Emptying, Hemostasis, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Ossification, Pathogenesis, Phagocytosis, Regeneration, Secretion, Transport, Transposition, Virulence, Wound Healing
Quite a number of genes have been found to play important roles in Wound Infection, such as ALB, ARSA, C2, CAT, CRAT, CRP, CSF2, CSRP1, GLYAT, GNL3, IL6, INS, KRAS, LAMC2, NDUFB6, SLC9A6, SS18L1, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.