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Disease Info Card
Hypervitaminosis D
Information about Hypervitaminosis D: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hypervitaminosis D
Most recent studies have shown that Hypervitaminosis D shares some biological mechanisms with arteriosclerosis, calcinosis, hypercalcemia, hypercalciuria, hyperphosphatemia, hypertensive-disease, hypervitaminosis, kidney-diseases, kidney-failure, kidney-failure-chronic, malignant-neoplasms, neoplasms, nephrocalcinosis, osteopenia, osteoporosis, poisoning-by-vitamin-d, rickets, vitamin-d-deficiency.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hypervitaminosis D, and have been seen in publications frequently: Aging, Bone Mineralization, Bone Resorption, Calcitonin Secretion, Cell Differentiation, Cell Proliferation, Diuresis, Excretion, Immune Response, Intestinal Absorption, Localization, Ossification, Pathogenesis, Pigmentation, Proteolysis, Response To Vitamin, Response To Vitamin D, Secretion, Transepithelial Transport, Transport
Quite a number of genes have been found to play important roles in Hypervitaminosis D, such as BGLAP, CALCA, CYP24A1, CYP27A1, CYP27B1, DCT, FGF23, IGF1, IGF1R, KL, POMC, PPARG, PTH, PTRH1, SPP1, TNF, TRPV5, TRPV6, VDR. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.