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Disease Info Card
Hypervitaminosis
Information about Hypervitaminosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hypervitaminosis
Most recent studies have shown that Hypervitaminosis shares some biological mechanisms with avitaminosis, bone-diseases, calcinosis, cleft-palate, dermatologic-disorders, fibrosis, hypercalcemia, hypertensive-disease, hypertrophy, hypervitaminosis-a, hypervitaminosis-d, kidney-failure, kidney-failure-chronic, neoplasms, osteoporosis, poisoning-by-vitamin-a, pseudotumor-cerebri, vitamin-a-deficiency, vitamin-deficiency.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hypervitaminosis, and have been seen in publications frequently: Aging, Bone Mineralization, Bone Resorption, Cell Differentiation, Cell Growth, Cell Proliferation, Endochondral Ossification, Excretion, Gluconeogenesis, Immune Response, Keratinization, Larval Development, Localization, Mitosis, Neural Tube Closure, Ossification, Pathogenesis, Retinol Transport, Secretion, Transport
Quite a number of genes have been found to play important roles in Hypervitaminosis, such as CALCA, CALR, CAT, CRAT, CYP27B1, DIO2, DIO3, FGF23, GLYAT, IGF1, KL, PTH, PTRH1, RBP4, RENBP, SUGP1, TRPV5, VDR. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.