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Disease Info Card
Hyperoxaluria
Information about Hyperoxaluria: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hyperoxaluria
Most recent studies have shown that Hyperoxaluria shares some biological mechanisms with cystinuria, hypercalciuria, hyperoxaluria-primary, inborn-errors-of-metabolism, intestinal-diseases, kidney-calculi, kidney-diseases, kidney-failure, kidney-failure-chronic, lithiasis, malabsorption-syndrome, metabolic-diseases, nephrocalcinosis, nephrolithiasis, obesity, primary-hyperoxaluria-type-i, urinary-tract-infection, urolithiasis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hyperoxaluria, and have been seen in publications frequently: Bone Resorption, Cell Death, Cell Proliferation, Coagulation, Diuresis, Donor Selection, Excretion, Glomerular Filtration, Gluconeogenesis, Inflammatory Response, Intestinal Absorption, Localization, Oxalate Transport, Pathogenesis, Protein Import, Reverse Transcription, Secretion, Translation, Transport, Urea Cycle
Quite a number of genes have been found to play important roles in Hyperoxaluria, such as AGT, AGXT, CAT, GRHPR, HAO1, HAO2, HCRT, PAH, PDXP, PHC1, PHC2, PLP1, PODXL2, PRDX5, PTH, PTHLH, RAPGEF5, SLC26A1, SPP1, UMOD. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.