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Disease Info Card
Hyperlipoproteinemia Type I
Information about Hyperlipoproteinemia Type I: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hyperlipoproteinemia Type I
Most recent studies have shown that Hyperlipoproteinemia Type I shares some biological mechanisms with acute-pancreatitis, arteriosclerosis, atherosclerosis, diabetes-mellitus, dyslipidemias, hypercholesterolemia, hypercholesterolemia-familial, hyperinsulinism, hyperlipidemia, hyperlipidemia-familial-combined, hyperlipoproteinemia-type-iib, hyperlipoproteinemia-type-iii, hyperlipoproteinemia-type-iv, hyperlipoproteinemia-type-v, hyperlipoproteinemias, insulin-resistance, obesity, pancreatitis, xanthoma.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hyperlipoproteinemia Type I, and have been seen in publications frequently: Coagulation, Excretion, Fibrinolysis, Gastric Emptying, Immune Response, Inflammatory Response, Insulin Secretion, Intestinal Absorption, Lactation, Lipid Binding, Lipoprotein Transport, Localization, Mrna Splicing, Pathogenesis, Phagocytosis, Platelet Aggregation, Secretion, Translation, Transport, Triglyceride Transport
Quite a number of genes have been found to play important roles in Hyperlipoproteinemia Type I, such as APOA1, APOA5, APOB, APOC2, APOC3, APOE, CD55, CETP, INS, LCP1, LDLR, LIPC, LPL, MCPH1, SLC16A1, TCN2, TG. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.