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Disease Info Card
Acute Pancreatitis
Information about Acute Pancreatitis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Acute Pancreatitis
Most recent studies have shown that Acute Pancreatitis shares some biological mechanisms with abdominal-pain, abscess, cholecystolithiasis, cholelithiasis, edema, hemorrhage, inflammation, malignant-neoplasms, multiple-organ-failure, necrosis-of-pancreas, neoplasms, pain, pancreatic-diseases, pancreatic-neoplasm, pancreatic-pseudocyst, pancreatitis, pancreatitis-acute-necrotizing, pancreatitis-chronic, systemic-infection.
Among the many pathways, these few ones have gauged particular interests from scientists studying Acute Pancreatitis, and have been seen in publications frequently: Autophagy, Blood Coagulation, Cell Death, Cell Proliferation, Coagulation, Complement Activation, Cytokine Production, Excretion, Fibrinolysis, Hemostasis, Immune Response, Inflammatory Response, Localization, Pathogenesis, Proteolysis, Regeneration, Secretion, Transport, Trypsinogen Activation, Zymogen Activation
Quite a number of genes have been found to play important roles in Acute Pancreatitis, such as ALB, APCS, CAT, CCK, CRP, CSRP1, IL10, IL6, INS, MPO, NFKB1, PLA2G1B, PLB1, RIPK2, SCT, SH2D1A, SST, TNF, YWHAZ. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.