Disease Info Card

Hyperchloraemia

Information about Hyperchloraemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Hyperchloraemia

Most recent studies have shown that Hyperchloraemia shares some biological mechanisms with acid-base-imbalance, acidosis, acidosis-hyperchloraemic, alkalosis, azotemia, bromide-poisoning, dehydration, diabetes-mellitus, hypernatremia, hypertensive-disease, hypochloraemia, kidney-diseases, kidney-failure, kidney-failure-chronic, malnutrition, metabolic-acidosis, poisoning, pseudohypoaldosteronism, renal-tubular-acidosis, water-electrolyte-imbalance.

Among the many pathways, these few ones have gauged particular interests from scientists studying Hyperchloraemia, and have been seen in publications frequently: Amino Acid Transport, Chloride Transport, Cognition, Diuresis, Excretion, Glomerular Filtration, Intestinal Absorption, Ion Transport, Menopause, Micturition, Muscle Atrophy, Oxygen Transport, Pathogenesis, Response To Drug, Secretion, Transport, Vasoconstriction, Vasodilation, Water Homeostasis, Wound Healing

Quite a number of genes have been found to play important roles in Hyperchloraemia, such as AGT, ALB, AVP, C2, CALB2, CASR, CD55, CR2, GNL3, INS, ITGA2, KRAS, PTH, PTRH1, RAPGEF5, REN, SLC12A3, WNK1, WNK4. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.