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Disease Info Card
Herlitz Disease
Information about Herlitz Disease: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Herlitz Disease
Most recent studies have shown that Herlitz Disease shares some biological mechanisms with atresia, bulla, cicatrix, dermatologic-disorders, edema, epidermolysis-bullosa, epidermolysis-bullosa-dystrophica, epidermolysis-bullosa-junctionalis-with-pyloric-atresia, fetal-diseases, hallopeau-siemens-disease, hypoplasia, junctional-epidermolysis-bullosa, nails-malformed, skin-diseases-genetic, skin-diseases-vesiculobullous, systemic-infection, tissue-adhesions.
Among the many pathways, these few ones have gauged particular interests from scientists studying Herlitz Disease, and have been seen in publications frequently: Hemidesmosome Assembly, Keratinization, Localization, Odontogenesis, Pathogenesis, Proteolysis, Secretion, Wound Healing
Quite a number of genes have been found to play important roles in Herlitz Disease, such as AMY2A, COL17A1, CTLA4, EPB42, FN1, GLB1, ITGA6, ITGB4, LAMA3, LAMA4, LAMB2, LAMB3, LAMC1, LAMC2, NOD2, PLEC, PLOD1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.
Pathways Related to Herlitz Disease
This information is being compiled and will come in a future update
| Hemidesmosome Assembly | Keratinization | Localization |
| Odontogenesis | Pathogenesis | Proteolysis |
| Secretion | Wound Healing |