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Disease Info Card
Granular Corneal Dystrophy
Information about Granular Corneal Dystrophy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Granular Corneal Dystrophy
Most recent studies have shown that Granular Corneal Dystrophy shares some biological mechanisms with amyloid-deposition, amyloidosis, avellino-corneal-dystrophy, cataract, corneal-deposits, corneal-diseases, dystrophy, familial-amyloid-polyneuropathy-type-v, hereditary-corneal-dystrophy, macular-corneal-dystrophy, plaque-amyloid, recurrence-(disease-attribute), reis-bucklers-corneal-dystrophy, stromal-corneal-dystrophy, visual-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Granular Corneal Dystrophy, and have been seen in publications frequently: Autophagy, Cell Adhesion, Cell Cycle, Cell Death, Cell Differentiation, Cell Growth, Integrin-mediated Signaling Pathway, Localization, Pathogenesis, Response To Oxidative Stress, Response To Wounding, Reverse Transcription, Secretion, Wound Healing
Quite a number of genes have been found to play important roles in Granular Corneal Dystrophy, such as ACD, BAX, BCL2L1, CAT, CHST6, GCDH, GLA, GSR, KRT12, KRT3, NAT8, NLGN1, PARP1, PTK2B, SMAD3, STS, TGFB1, TGFBI. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.