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- Table of Contents
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Disease Info Card
Gait, Drop Foot
Information about Gait, Drop Foot: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Gait, Drop Foot
Most recent studies have shown that Gait, Drop Foot shares some biological mechanisms with asthma, cerebral-palsy, cerebrovascular-accident, decerebrate-state, depressive-disorder, flexed-fetal-attitude, gait-disorders-neurologic, impairment-(finding), malignant-neoplasms, malnutrition, neoplasms, nervousness, pain, paraplegia, parkinson-disease, secondary-parkinson-disease, spinal-cord-injuries, spinal-cord-transection-injury, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Gait, Drop Foot, and have been seen in publications frequently: Aging, Cognition, Flight, Forward Locomotion, Inflammatory Response, Innervation, Intracellular Transport, Localization, Locomotion, Locomotor Rhythm, Muscle Contraction, Neuroprotection, Pathogenesis, Proprioception, Reflex, Regeneration, Secretion, Swimming, Translation, Transport
Quite a number of genes have been found to play important roles in Gait, Drop Foot, such as BDNF, C2, CARD16, CAT, CP, CRAT, FES, GATA3, GHRH, GLYAT, HNRNPC, MYH14, MYO5A, MYO6, NR3C2, SLC17A5, TFF2, TH, TNFSF10. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.