Disease Info Card

Spinal Cord Transection Injury

Information about Spinal Cord Transection Injury: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Spinal Cord Transection Injury

Most recent studies have shown that Spinal Cord Transection Injury shares some biological mechanisms with atrophy, autonomic-dysreflexia, bradycardia, contusions, decerebrate-state, disease-of-adrenal-medulla, flexed-fetal-attitude, gait-drop-foot, hypertensive-disease, hypotension-adverse-event, muscle-spasticity, muscular-atrophy, nervousness, neurogenic-urinary-bladder, pain, paraplegia, pathological-dilatation, quadriplegia, spinal-cord-injuries, spinal-injuries.

Among the many pathways, these few ones have gauged particular interests from scientists studying Spinal Cord Transection Injury, and have been seen in publications frequently: Axon Regeneration, Cell Death, Cell Proliferation, Excretion, Gastric Emptying, Innervation, Localization, Locomotion, Micturition, Muscle Atrophy, Neurogenesis, Neuroprotection, Pathogenesis, Reflex, Regeneration, Secretion, Swimming, Synaptic Transmission, Transport, Vasoconstriction

Quite a number of genes have been found to play important roles in Spinal Cord Transection Injury, such as ARHGAP4, BDNF, C2, CALCA, CAT, CD38, CHAT, CRAT, FGF1, GFAP, GLYAT, HNRNPC, NTF3, PFDN4, POMC, PRSS3, RPL6, TAC1, TFRC, TH. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.