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Disease Info Card
Ossifying Fibroma
Information about Ossifying Fibroma: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Ossifying Fibroma
Most recent studies have shown that Ossifying Fibroma shares some biological mechanisms with bone-neoplasms, cementoma, dysplasia, fibroma, fibrous-dysplasia, gingival-neoplasms, heterotopic-ossification, jaw-neoplasms, juvenile-ossifying-fibroma, malignant-paraganglionic-neoplasm, mandibular-neoplasms, maxillary-neoplasms, neoplasm-recurrence-local, neoplasms, odontogenic-tumors, osteitis-fibrosa-disseminata, osteoma, paranasal-sinus-neoplasms, skull-neoplasms.
Among the many pathways, these few ones have gauged particular interests from scientists studying Ossifying Fibroma, and have been seen in publications frequently: Bone Maturation, Bone Remodeling, Cell Cycle, Cell Differentiation, Cell Proliferation, Endochondral Ossification, Enucleation, Keratinization, Localization, Mastication, Mitosis, Oncogenesis, Ossification, Osteoblast Differentiation, Osteoclast Differentiation, Pathogenesis, Regeneration, Tooth Eruption, Transport
Quite a number of genes have been found to play important roles in Ossifying Fibroma, such as BGLAP, CDC73, DES, DIAPH2, ENO2, ETFA, FMR1, GFAP, GNAS, MUC1, NCAM1, PCNA, S100A1, S100B, SPP1, TNFRSF11A, TNFRSF11B, VIM. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.