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Disease Info Card
Fatal Familial Insomnia
Information about Fatal Familial Insomnia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Fatal Familial Insomnia
Most recent studies have shown that Fatal Familial Insomnia shares some biological mechanisms with ataxia, atrophy, creutzfeldt-jakob-disease, creutzfeldt-jakob-disease-familial, dementia, dysautonomia, encephalopathies, encephalopathy-bovine-spongiform, gerstmann-straussler-scheinker-disease, gliosis, kuru, neurodegenerative-disorders, new-variant-creutzfeldt-jakob-disease, pain, prion-diseases, scrapie, sleep-disorders, sleep-initiation-and-maintenance-disorders, sleeplessness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Fatal Familial Insomnia, and have been seen in publications frequently: Aging, Apoptotic Process, Cell Death, Cell Division, Cellular Homeostasis, Circadian Rhythm, Glycosylation, Immune Response, Mating, Menstruation, Pathogenesis, Phagocytosis, Programmed Cell Death, Secretion, Secretory Pathway, Swimming, Translation, Transport, Tropism, Virulence
Quite a number of genes have been found to play important roles in Fatal Familial Insomnia, such as ABCB6, C4BPA, CARD14, CSF2, F2R, GSS, LAMC2, MAPT, PLN, PRDX2, PRNP, TFRC, TMEFF2, TXNRD1, TXNRD2, YWHAQ. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.