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Disease Info Card
Creutzfeldt-jakob Disease
Information about Creutzfeldt-jakob Disease: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Creutzfeldt-jakob Disease
Most recent studies have shown that Creutzfeldt-jakob Disease shares some biological mechanisms with alzheimers-disease, ataxia, atrophy, brain-diseases, cns-disorder, dementia, encephalopathies, encephalopathy-bovine-spongiform, gerstmann-straussler-scheinker-disease, gliosis, infective-disorder, kuru, myoclonus, nervousness, neurodegenerative-disorders, new-variant-creutzfeldt-jakob-disease, prion-diseases, scrapie, sporadic-cjd.
Among the many pathways, these few ones have gauged particular interests from scientists studying Creutzfeldt-jakob Disease, and have been seen in publications frequently: Aging, Amyloid Fibril Formation, Autophagy, Cell Death, Coagulation, Cognition, Donor Selection, Glycosylation, Immune Response, Inflammatory Response, Localization, Pathogenesis, Programmed Cell Death, Protein Folding, Proteolysis, Reflex, Secretion, Translation, Transport, Virulence
Quite a number of genes have been found to play important roles in Creutzfeldt-jakob Disease, such as ABCB6, APP, C4BPA, CARD14, CSF2, ENO1, ENO2, GFAP, GSS, LAMC2, MAPT, NLRP5, PRDX2, PRND, PRNP, TH, YWHAQ. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.