Disease Info Card

Fasting Hypoglycemia

Information about Fasting Hypoglycemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Fasting Hypoglycemia

Most recent studies have shown that Fasting Hypoglycemia shares some biological mechanisms with acidosis, adenoma, autoimmune-reaction, diabetes-mellitus, glycogen-storage-disease, glycogen-storage-disease-type-i, hepatomegaly, hyperglycemia, hyperinsulinism, hypoglycemia, insulin-resistance, insulin-sensitivity, insulinoma, islet-cell-adenoma, ketosis, neoplasms, obesity, pancreatic-neoplasm, reactive-hypoglycemia, storage-disease.

Among the many pathways, these few ones have gauged particular interests from scientists studying Fasting Hypoglycemia, and have been seen in publications frequently: Enucleation, Excretion, Fatty Acid Oxidation, Glucagon Secretion, Gluconeogenesis, Glucose Homeostasis, Glucose Transport, Glycolysis, Hormone Secretion, Hypersensitivity, Insulin Secretion, Lipid Oxidation, Localization, Membrane Depolarization, Pathogenesis, Regeneration, Regulation Of Gluconeogenesis, Secretion, Transport, Urea Cycle

Quite a number of genes have been found to play important roles in Fasting Hypoglycemia, such as ABCC8, AKAP4, G6PC, GCG, GGH, GH1, GLUD1, H6PD, IGF1, IGF2, IGFBP1, IGFBP3, INS, INSR, POMC, SLC2A2, SLC37A4, SST. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.