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- Table of Contents
Information about Eosinophilic Pneumonia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Eosinophilic Pneumonia shares some biological mechanisms with asthma, bronchiolitis, carringtons-pulmonary-eosinophilia, chronic-eosinophilic-pneumonia, churg-strauss-syndrome, coughing, dyspnea, eosinophilia, fibrosis, inflammation, loffler-syndrome, lung-diseases, organising-pneumonia, pleural-effusion-disorder, pneumonia, porphyria-erythropoietic, pulmonary-eosinophilia, pulmonary-fibrosis, respiratory-failure, vasculitis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Eosinophilic Pneumonia, and have been seen in publications frequently: Cell Activation, Cell Adhesion, Chemotaxis, Coagulation, Cytokine Production, Eosinophil Activation, Eosinophil Chemotaxis, Eosinophil Degranulation, Eosinophil Migration, Fertilization, Hypersensitivity, Immune Response, Inflammatory Response, Leukocyte Migration, Pathogenesis, Phagocytosis, Proteolysis, Secretion, Sensitization, Transport
Quite a number of genes have been found to play important roles in Eosinophilic Pneumonia, such as CCL11, CCL17, CCL22, CD4, CD8A, CRP, CSF2, CTLA4, DBI, EPO, IL10, IL13, IL2, IL3, IL5, LGMN, MUC1, RNASE3, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.