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Disease Info Card
Churg-strauss Syndrome
Information about Churg-strauss Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Churg-strauss Syndrome
Most recent studies have shown that Churg-strauss Syndrome shares some biological mechanisms with anti-neutrophil-cytoplasmic-antibody-associated-vasculitis, arteritis, asthma, eosinophilia, glomerulonephritis, granuloma, hemorrhage, inflammation, lung-diseases, microscopic-polyangiitis, necrotizing-vasculitis, pneumonia, polyarteritis-nodosa, purpura, vasculitis, wegener-granulomatosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Churg-strauss Syndrome, and have been seen in publications frequently: Angiogenesis, Blood Coagulation, Cell Activation, Cell Adhesion, Coagulation, Cytokine Production, Eosinophil Activation, Excretion, Glomerular Filtration, Granuloma Formation, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Neutrophil Activation, Pathogenesis, Respiratory Burst, Secretion, Sensitization, T Cell Activation
Quite a number of genes have been found to play important roles in Churg-strauss Syndrome, such as ACR, AGA, CD4, CMAS, CRP, CSRP1, CYCS, GNAI1, IFNA1, IL10, IL13, IL2, IL4, IL5, IL6, MPO, PRTN3, RNASE3, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.