Disease Info Card

Pulmonary Eosinophilia

Information about Pulmonary Eosinophilia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Pulmonary Eosinophilia

Most recent studies have shown that Pulmonary Eosinophilia shares some biological mechanisms with allergic-asthma, allergy, asthma, bronchial-hyperreactivity, chronic-eosinophilic-pneumonia, coughing, dyspnea, eosinophilia, eosinophilic-pneumonia, fibrosis, filariasis, inflammation, loffler-syndrome, lung-diseases, lung-infiltration, pleural-effusion-disorder, pneumonia, pulmonary-fibrosis, respiratory-hypersensitivity, tropical-eosinophilia.

Among the many pathways, these few ones have gauged particular interests from scientists studying Pulmonary Eosinophilia, and have been seen in publications frequently: Cell Activation, Cell Adhesion, Cell Differentiation, Cell Migration, Cell Proliferation, Chemotaxis, Cytokine Production, Cytokine Secretion, Eosinophil Activation, Eosinophil Chemotaxis, Eosinophil Degranulation, Eosinophil Migration, Hypersensitivity, Immune Response, Inflammatory Response, Mast Cell Degranulation, Pathogenesis, Secretion, Sensitization, T Cell Activation

Quite a number of genes have been found to play important roles in Pulmonary Eosinophilia, such as AHR, CCL11, CCL5, CD4, CD8A, CSF2, CTLA4, EPO, EPX, IFNG, IL10, IL13, IL2, IL4, IL5, IL6, RNASE3, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.