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Disease Info Card
Transposition Of Great Vessels
Information about Transposition Of Great Vessels: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Transposition Of Great Vessels
Most recent studies have shown that Transposition Of Great Vessels shares some biological mechanisms with aortic-coarctation, atresia, cardiac-arrhythmia, congenital-heart-defects, congenital-heart-disease, corrected-transposition-of-great-vessels, double-outlet-right-ventricle, heart-diseases, heart-failure, heart-septal-defects, patent-ductus-arteriosus, pulmonary-stenosis, pulmonary-valve-stenosis, regurgitation, situs-inversus, stenosis, tetralogy-of-fallot, ventricular-septal-defects.
Among the many pathways, these few ones have gauged particular interests from scientists studying Transposition Of Great Vessels, and have been seen in publications frequently: Angiogenesis, Brain Development, Coagulation, Cognition, Dehiscence, Excretion, Hemostasis, Inflammatory Response, Localization, Mating, Ossification, Pathogenesis, Response To Stress, Secretion, Segmentation, Translation, Transport, Transposition, Vasoconstriction, Vasodilation
Quite a number of genes have been found to play important roles in Transposition Of Great Vessels, such as AMY2A, BCHE, BLOC1S6, CELA3B, CPB1, ENOPH1, EPB42, GDF1, GNL3, IL10, IL6, KRAS, PRH1, SS18L1, TBX1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.