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Disease Info Card
Double Outlet Right Ventricle
Information about Double Outlet Right Ventricle: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Double Outlet Right Ventricle
Most recent studies have shown that Double Outlet Right Ventricle shares some biological mechanisms with aortic-coarctation, atresia, atrial-septal-defects, common-ventricle-disorder, congenital-atresia-of-pulmonary-valve, congenital-heart-defects, congenital-heart-disease, endocardial-cushion-defects, heart-diseases, heart-septal-defects, hypoplasia, pulmonary-stenosis, pulmonary-valve-stenosis, regurgitation, stenosis, tetralogy-of-fallot, transposition-of-great-vessels, truncus-arteriosus-persistent, ventricular-septal-defects.
Among the many pathways, these few ones have gauged particular interests from scientists studying Double Outlet Right Ventricle, and have been seen in publications frequently: Angiogenesis, Artery Development, Catecholamine Secretion, Cell Death, Cell Migration, Coagulation, Dehiscence, Developmental Process, Heart Development, Innervation, Localization, Mating, Neural Crest Cell Migration, Neural Tube Closure, Ossification, Outflow Tract Morphogenesis, Pathogenesis, Secretion, Transposition, Tube Closure
Quite a number of genes have been found to play important roles in Double Outlet Right Ventricle, such as AMY2A, BLOC1S6, BMP4, CPB1, EPB42, FEZF2, GDF1, NKX2-5, PAX3, PITX2, PRH1, SLC17A5, SS18L1, TBX1, VEGFA, ZFPM2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.