Ehlers-danlos Syndrome, Type 4

Overview of Ehlers-danlos Syndrome, Type 4

Most recent studies have shown that Ehlers-danlos Syndrome, Type 4 shares some biological mechanisms with aneurysm, aneurysm-dissecting, aortic-aneurysm, aortic-rupture, connective-tissue-diseases, contusions, ehlers-danlos-syndrome, hemorrhage, hereditary-diseases, intestinal-perforation, intracranial-aneurysm, marfan-syndrome, pathologic-fistula, pathological-dilatation, perforation-of-colon, pregnancy-complications, rupture-of-artery, rupture-spontaneous, vascular-diseases.

Among the many pathways, these few ones have gauged particular interests from scientists studying Ehlers-danlos Syndrome, Type 4, and have been seen in publications frequently: Coagulation, Gastric Emptying, Glycosylation, Localization, Mrna Splicing, Pathogenesis, Platelet Aggregation, Response To Stress, Rna Splicing, Secretion, Wound Healing

Quite a number of genes have been found to play important roles in Ehlers-danlos Syndrome, Type 4, such as BGN, CD63, COL1A1, COL1A2, COL2A1, COL3A1, CTLA4, ELANE, ELN, FBN1, ICA, LOX, PGA4, PGC, PSMD1, RET, RNF7, TGFBR2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Ehlers-danlos Syndrome, Type 4 Related Genes

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Pathways Related to Ehlers-danlos Syndrome, Type 4

This information is being compiled and will come in a future update

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