Disease Info Card

Corticobasal Degeneration

Information about Corticobasal Degeneration: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Corticobasal Degeneration

Most recent studies have shown that Corticobasal Degeneration shares some biological mechanisms with abnormal-degeneration, alzheimers-disease, aphasia, apraxias, atrophy, basal-ganglia-diseases, dementia, frontotemporal-dementia, lewy-body-disease, movement-disorders, multiple-system-atrophy, nerve-degeneration, neurodegenerative-disorders, neurofibrillary-tangles, parkinson-disease, pick-disease-of-the-brain, progressive-supranuclear-palsy, secondary-parkinson-disease, supranuclear-palsy, tauopathies.

Among the many pathways, these few ones have gauged particular interests from scientists studying Corticobasal Degeneration, and have been seen in publications frequently: Aging, Cell Cycle, Cell Death, Cognition, Hyperphosphorylation, Hypersensitivity, Innervation, Localization, Mrna Splicing, Muscle Contraction, Neuroprotection, Pathogenesis, Phagocytosis, Programmed Cell Death, Proteolysis, Reflex, Segmentation, Translation, Transport, Visual Perception

Quite a number of genes have been found to play important roles in Corticobasal Degeneration, such as APOE, BPIFA2, CBS, COPS2, CSF2, GFAP, GRN, LAMC2, MAPT, MSMB, PRNP, PSPH, PSPN, REG1A, SNCA, SOD1, STXBP3, TARDBP, TPO. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.