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Disease Info Card
Congenital Pain Insensitivity
Information about Congenital Pain Insensitivity: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Congenital Pain Insensitivity
Most recent studies have shown that Congenital Pain Insensitivity shares some biological mechanisms with anhidrosis, arthropathy, congenital-abnormality, dysautonomia-familial, fracture, hereditary-sensory-and-autonomic-neuropathies, hsan-type-iv, hypohidrosis, indifferent-mood, neurogenic-arthropathy, osteomyelitis, pain, sensory-neuropathy, sensory-neuropathy-hereditary, ulcer.
Among the many pathways, these few ones have gauged particular interests from scientists studying Congenital Pain Insensitivity, and have been seen in publications frequently: Cell Death, Chemotaxis, Excretion, Immune Response, Inflammatory Response, Innervation, Insulin Secretion, Localization, Mastication, Operant Conditioning, Pancreas Morphogenesis, Pathogenesis, Programmed Cell Death, Reflex, Response To Pain, Sensory Processing, Signal Transmission, Swimming, Tooth Eruption, Translation
Quite a number of genes have been found to play important roles in Congenital Pain Insensitivity, such as BDNF, CALCA, CSF2, KCNIP3, LAMC2, MTSS1, NAA50, NGF, NGFR, NTRK1, POMC, PSEN2, SCN10A, SCN3A, SCN9A, SPTLC1, TAC1, TAF9. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.