This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Hsan Type Iv
Information about Hsan Type Iv: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hsan Type Iv
Most recent studies have shown that Hsan Type Iv shares some biological mechanisms with anhidrosis, cicatrix, congenital-pain-insensitivity, dysautonomia-familial, fracture, hereditary-diseases, hereditary-sensory-and-autonomic-neuropathies, hyperpyrexia, hypohidrosis, infective-disorder, nervousness, osteomyelitis, pain, sensory-neuropathy, sensory-neuropathy-hereditary, ulcer.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hsan Type Iv, and have been seen in publications frequently: Biological Regulation, Chemotaxis, Chronic Inflammatory Response, Circadian Rhythm, Habituation, Inflammatory Response, Innervation, Insulin Secretion, Intracellular Signal Transduction, Localization, Locomotion, Mrna Splicing, Pancreas Morphogenesis, Pathogenesis, Proprioception, Reflex, Response To Pain, Response To Stress, Sensory Perception, Wound Healing
Quite a number of genes have been found to play important roles in Hsan Type Iv, such as ARTN, BCL2L1, BDNF, CSF2, INS, ITCH, LAMC2, MAPK3, MIF, NAA50, NGF, NGFR, NTF3, NTRK1, SPTLC1, SS18L1, TAC1, TPM3. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.