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Disease Info Card
Congenital Nephrogenic Diabetes Insipidus
Information about Congenital Nephrogenic Diabetes Insipidus: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Congenital Nephrogenic Diabetes Insipidus
Most recent studies have shown that Congenital Nephrogenic Diabetes Insipidus shares some biological mechanisms with acidosis, bartter-disease, central-diabetes-insipidus, dehydration, deprivation-of-water, diabetes-mellitus, failure-to-thrive, glycosuria-renal, hereditary-diseases, hydronephrosis, hypernatremia, kidney-diseases, nephrogenic-diabetes-insipidus, pituitary-diseases, polydipsia, polyuria, pregnancy-complications, renal-tubular-acidosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Congenital Nephrogenic Diabetes Insipidus, and have been seen in publications frequently: Coagulation, Diuresis, Excretion, Exocytosis, Glomerular Filtration, Hormone Secretion, Localization, Methylation, Milk Ejection, Natriuresis, Pathogenesis, Platelet Aggregation, Response To Water Deprivation, Secretion, Translation, Transport, Vasoconstriction, Vasodilation, Water Homeostasis, Water Transport
Quite a number of genes have been found to play important roles in Congenital Nephrogenic Diabetes Insipidus, such as APC, AQP1, AQP2, AVP, AVPR2, DBT, GLUL, IFNAR1, IGKV1-5, NLRP3, PLAT, PPOX, PTH, REN, SLC4A1, VWF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.