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Disease Info Card
Chronic Rejection Of Renal Transplant
Information about Chronic Rejection Of Renal Transplant: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Chronic Rejection Of Renal Transplant
Most recent studies have shown that Chronic Rejection Of Renal Transplant shares some biological mechanisms with atrophy, decreased-immunologic-activity-[pe], delayed-graft-function, diabetes-mellitus, fibrosis, glomerulonephritis, glomerulosclerosis-(disorder), graft-failure, hypertensive-disease, infective-disorder, inflammation, interstitial-fibrosis, ischemia, kidney-diseases, kidney-failure, kidney-failure-chronic, proteinuria-of-undiagnosed-cause, renal-glomerular-disease.
Among the many pathways, these few ones have gauged particular interests from scientists studying Chronic Rejection Of Renal Transplant, and have been seen in publications frequently: Aging, Cell Activation, Cell Adhesion, Cell Cycle, Cell Proliferation, Diuresis, Excretion, Glomerular Filtration, Immune Response, Inflammatory Response, Localization, Lymphocyte Proliferation, Mesenchymal Cell Proliferation, Pathogenesis, Regeneration, Secretion, Senescence, Sensitization, Vasoconstriction, Wound Healing
Quite a number of genes have been found to play important roles in Chronic Rejection Of Renal Transplant, such as ACE, AGT, AKR1B1, AR, AREG, CABIN1, CCL2, FDXR, GNL3, IL10, IL2, IL6, KRAS, MTOR, RAPGEF5, SRL, TGFB1, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.