Disease Info Card

Chronic Graft Versus Host Disease

Information about Chronic Graft Versus Host Disease: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Chronic Graft Versus Host Disease

Most recent studies have shown that Chronic Graft Versus Host Disease shares some biological mechanisms with acute-gvh-disease, acute-lymphocytic-leukemia, anemia, aplastic-anemia, autoimmune-reaction, cytomegalovirus-infections, decreased-immunologic-activity-[pe], dysmyelopoietic-syndromes, graft-failure, graft-vs-host-disease, hematologic-neoplasms, infective-disorder, leukemia, leukemia-myelocytic-acute, lymphoma, malignant-neoplasms, myeloid-leukemia, myeloid-leukemia-chronic, neoplasms, pneumonia.

Among the many pathways, these few ones have gauged particular interests from scientists studying Chronic Graft Versus Host Disease, and have been seen in publications frequently: B Cell Activation, Cell Activation, Cell Adhesion, Cell Death, Cell Differentiation, Cell Proliferation, Cytokine Production, Cytokine Secretion, Donor Selection, Excretion, Hypersensitivity, Immune Response, Localization, Pathogenesis, Pigmentation, Regeneration, Secretion, Sensitization, T Cell Activation, Tolerance Induction

Quite a number of genes have been found to play important roles in Chronic Graft Versus Host Disease, such as BCR, CD34, CD4, CD8A, CSF3, CTLA4, EFS, FXYD5, HLA-A, HLA-DQA1, HLA-DRB4, IFNG, IL10, IL2, MID1, NOD2, NRM, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.