Disease Info Card

Acute Gvh Disease

Information about Acute Gvh Disease: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Acute Gvh Disease

Most recent studies have shown that Acute Gvh Disease shares some biological mechanisms with acute-leukemia, acute-lymphocytic-leukemia, anemia, aplastic-anemia, chronic-graft-versus-host-disease, cytomegalovirus-infections, decreased-immunologic-activity-[pe], dysmyelopoietic-syndromes, graft-failure, graft-vs-host-disease, hematologic-neoplasms, infective-disorder, leukemia, leukemia-myelocytic-acute, lymphoma, malignant-neoplasms, myeloid-leukemia, myeloid-leukemia-chronic, neoplasms, pneumonia.

Among the many pathways, these few ones have gauged particular interests from scientists studying Acute Gvh Disease, and have been seen in publications frequently: Cell Activation, Cell Adhesion, Cell Death, Cell Differentiation, Cell Proliferation, Cytokine Production, Cytokine Secretion, Donor Selection, Excretion, Glomerular Filtration, Hypersensitivity, Immune Response, Inflammatory Response, Lymphocyte Proliferation, Pathogenesis, Regeneration, Reverse Transcription, Secretion, T Cell Activation, T Cell Proliferation

Quite a number of genes have been found to play important roles in Acute Gvh Disease, such as BCR, CD34, CD4, CD8A, CSF3, CTLA4, FXYD5, HLA-A, HLA-DQA1, HLA-DRB4, IFNG, IL10, IL2, IL4, IL6, MID1, NOD2, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.