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Disease Info Card
Burkholderia Infections
Information about Burkholderia Infections: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Burkholderia Infections
Most recent studies have shown that Burkholderia Infections shares some biological mechanisms with abscess, bacteremia, basal-cell-carcinoma, cross-infection, cystic-fibrosis, fibrosis, granulomatous-disease-chronic, granulomatous-disorder, infectious-lung-disorder, infective-disorder, inflammation, lung-diseases, melioidosis, opportunistic-infections, pneumonia, pneumonia-bacterial, pseudomonas-infections, respiratory-tract-infections, systemic-infection.
Among the many pathways, these few ones have gauged particular interests from scientists studying Burkholderia Infections, and have been seen in publications frequently: Autophagy, Biofilm Formation, Cell Death, Chemotaxis, Colony Morphology, Drug Resistance, Host-pathogen Interaction, Humoral Immune Response, Immune Response, Inflammatory Response, Innate Immune Response, Localization, Methylation, Pathogenesis, Phagocytosis, Quorum Sensing, Secretion, Swimming, Transport, Virulence
Quite a number of genes have been found to play important roles in Burkholderia Infections, such as AMY2A, BLOC1S6, CCL2, CFTR, CRP, CYBB, EPB42, FEV, IL6, MBL2, MMAA, NCF1, PLXNA1, PRH1, RAD51, RPL10, TLR4, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.