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Disease Info Card
Opportunistic Infections
Information about Opportunistic Infections: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Opportunistic Infections
Most recent studies have shown that Opportunistic Infections shares some biological mechanisms with acquired-immunodeficiency-syndrome, aids-related-opportunistic-infections, bacterial-infections, candidiasis, cytomegalovirus-infections, decreased-immunologic-activity-[pe], hiv-infections, immunologic-deficiency-syndromes, infective-disorder, kaposi-sarcoma, leukemia, lymphoma, malignant-neoplasms, mycoses, neoplasms, pneumonia, pneumonia-pneumocystis-carinii, sarcoma, tuberculosis, virus-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying Opportunistic Infections, and have been seen in publications frequently: Aging, Cell Activation, Cell Death, Cell Proliferation, Coagulation, Cytokine Production, Drug Resistance, Excretion, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Lymphocyte Proliferation, Pathogenesis, Phagocytosis, Secretion, Transport, Tropism, Viral Replication, Virulence
Quite a number of genes have been found to play important roles in Opportunistic Infections, such as ARC, CD4, CD8A, CHP1, CRP, CSF2, CSF3, CTLA4, HLA-DQA1, IFNG, IL10, IL2, IL6, LAMC2, NOD2, NOL3, NRSN1, TMED2, TNF, TPPP. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.